An article search of dental literature up
to May 2010 in the databank PubMed using the keywords “bond strength”
and “dentin” or “enamel” yielded 2286 publications about adhesive bond
testing on dentin and 1360 about the same on enamel. In addition, 907
publications are listed in which the dye penetration test was used
(search words “microleakage” and “dentin”). Selected articles were
compared in terms of their test protocol and critically discussed in the
context of the existing literature. A comparison of laboratory data
with the results of clinical tests of adhesives will show the relevance
of the in vitro test methods
دانلود ترجمه مقاله استفاده از سایتوکین به عنوان یک بزاق در غربالکری اسکواموس سل کارسینوما دهان
قسمتی از متن انگلیسی:
The use of saliva for diagnosis of disease
has been sought centuries ago. However, the introduction of molecular
laboratory techniques in 1980s has drawn more attention to saliva as a
tool for detection of disease and for health monitoring in general.[23]
Oral fluid analysis has been proved useful for detection of Sjögren’s
syndrome,[24,25] diabetes mellitus,[26] infection with Helicobacter
pylori, [27] Cushing syndrome,[28] HIV,[29-31] and hepatitis C
virus.[32,33] It is now being routinely used for the detection of abused
as well as therapeutic drugs.[20] Saliva has also been suggested for
breast cancer screening programs by detecting Her2\neu.[34-36] Withal,
diseases of the oral cavity have been the most attractive ones for
saliva diagnostics researchers, and the use of saliva for detection of
dental caries[37] and periodontal diseases[38,39] has been suggested.
Salivary diagnostics for OSCC, if becoming a routine, would also
comprise a suitable tool for population screening, monitoring of
patients at risk of recurrent tumor, and consequently for improving the
survival rate of patients with this disease.[23,40]
دانلود مقاله انگلیسی با ترجمه بیماری پریودنتال، مکانیسم های التهابی آنالیز استخوان
قسمتی از متن انگلیسی:
۲٫ Resident Cells and Innate Immunity
The resident cells involved in the innate
host response are many including epithelial cells, gingival and
periodontal ligament (PDL) fibroblasts, osteoblast, and dendritic cells
[9]. Epithelial cells produce interleukin-8 (IL-8), a neutrophil
chemoattractant, which recruits neutrophils migration [13] and increases
monocyte adhesion in the blood vessels. Neutrophils that enter the
periodontal environment are primed and exhibit increased production of
proinflammatory cytokines such as interleukin-1 (IL-1), interleukin-6
(IL-6), and tumor necrosis factor-𝛼 (TNF-𝛼) [۱۴]. These cytokines
mediate periodontal tissue destruction by stimulating bone resorption.
Monocytes, on the other hand, can differentiate into osteoclasts (OCs)
upon different triggers while producing inflammatory cytokines as well;
expression of Wnt5a was recently reported in response to
lipopolysaccharide (LPS) [15].